Schizophrenia: A Proposed Cause and Cure
by David Barnes
back
Townsend Letter for Doctors, June, 1994, #131
Schizophrenia has traditionally been treated as a "reactive" psychiatric
disorder, i.e., "Logical but maladaptive reactions to common life
events." Sigmund Freud, however, "
concluded that some aspects
of schizophrenia were beyond a purely psychological understanding."
This paper will expound on and validate that shrewd observation
of the early century. It will present the argument for the genesis
of schizophrenia being of the "process" or chronic type caused
by a physical infection whose secretions provoke a chemically
induced biological reaction.
What is our point of departure? Let us review what the possible
infectious microbes are in very liberal and broad terms that are
really old classifications of the Five Kingdoms defined by biologists.
We note that biologists claim there are the Five Kingdoms (some
say a further division is needed claiming Six Kingdoms). Neither
the Five nor the Six include the viruses. Biologists deal with
living things and viruses are dead. Viruses are dealt with as
an addendum because viruses affect living cells. Doctors' needs
are different: their laboratory is the living human body: their
patient.
Doctors' perspectives are best suited with only three major divisions
of infectious microbes which must include viruses. No less and
no more than three. Each has infinite branching to satisfy those
who want variety. Those three simplified branches of infectious
microbes will be: the Viruses, the Bacteria and Fungi.
Viruses are dead DNA/RNA programs that go active when they pierce
a living cell. We will accept the biologist's definitions. These
viruses are lifeless cell takeover programs that use the nucleus
of the host's cell (its yolk?) to replicate their viral DNA/RNA
codes into new viral clones. They can be hot or cold infestations
(feverish or non-feverish). They can be pan systemic or a singular,
well-defined tumor, i.e., wart. They function best in an unlimited,
direct blood supply. Some organize a very intense venous system
or a tuber-like root structure for blood nourishment if they are
not swimming free in the blood, i.e., hematogenous. The blood
is really just another body organ like the brain, kidney, or like
its closest relative, the liver. The body does a remarkable job
of defending itself in a week to ten days for many of these viruses.
A defensive fever is commonly associated with these infections.
"Cooking" the virus is one of the body's major weapons for combating
a virus. Somehow the body being chilled below normal temperature
seems to make the patient much more susceptible to a new viral
infection and the recurrent fever blisters, Herpes #1/#2. Aspirin
is the universal anti-viral chemical. Aspirin is the best chemical
to eliminate warts, (Verruca Vulgaris) and is given in a hard
skin penetrating solvent (ether). Aspirin is given for all cold
or flu symptoms of viral origin, with bed rest. A less noted potassium
ion (K) and iodine seem to both be antiviral. The iodine and potassium
are agents that I think can help the human body's anti-viral armory.
This remotely implies the possible use of lithium ion usage as
a helper as well as the halogens other than iodine. Lighter and
more active members of the same Periodic Chart column replace
the heavier atoms. Sodium is used for nerve transmission and Li
would displace it altering synapse transfer and alter things like
the Hebe-gebes (mania, puberty).
An auto or serum inoculation (internal body defense or injected/oral)
is a brilliant method to prevent or subdue a viral infection in
the future no matter how intense that particular virus' assault.
This is why a cold/flu vaccine is always being modified: the old
one is now rendered inert by those that had it or one extremely
close to its configuration. Exceptions to this are found: an example
noted is the herpes #1 or #2 which are recurrent. The bacteria
are really animals of a very primitive sort. The classic definition
of an animal is that it is an organism that produces a form of
metabolized nitrogen as urea which it can no longer use. For the
sake of brevity, we will include those microbes that biologists
will classify as other than Monera. These are Protsita and leave
the door open to any primitive, Micro-Animilia. Intestinal worms
(Nemathelminthes and Platythelminthes but Annelida doesn't seem
to be important) are curiously treated with an anti-fungal because
they often have a fungus in their digestive tract. Examples of
anti-fungal agents are the tri-cyclenes and nicotine. All the
bacteria should be called fauna and treated as such with their
own individual, minor idiosyncrasies.
The more primitive the bacteria the more closely it resembles
the primitive fungi. Blue-green algae are a bit of both bacteria
and fungi. It's like a fungus that has learned to exist in a saline
solution and a bacteria that can make chlorophyll. The differentiating
attributes of bacteria (fauna) from fungi (flora) is the use of
B12 in place of the fungi (flora) chlorophyll, which in primitive
bacteria and fungi are still interchangeable. When we try to stop
or reduce the nutrients for a fungal growth by minimizing the
intake of chlorophyll, we must also limit the intake of vitamin
B-12, its look-alike with the center atom changed to cobalt. Fungi
can do well in a very cold environment: bacteria become inert
when below 40°F. Note the delicate effects temperature ranges
of only a few degrees Fahrenheit have on animal life forms. We
really have a blood temperature of 526°F above absolute zero,
a 1% change makes us sick, and a 10% reduction stops bacterial
growth. Fungi do well even with another 10% reduction. The exceptions
are that some bacteria stay alive up to 838° absolute temperature
in dry heat.
Since the bacteria are animals, they can live in the bloodstream
just like the virus. With that fantastic food supply they can
propagate very rapidly while secreting their toxins. This fast
cell division means they are a high heat liberating reaction or
a rapid metabolism. I don't think this is a measurable rise, I
think the body uses other reactions to generate the associated
fever. The body's defense must be quick and intense to be a good
defense against bacteria. A lot of fungi don't make a difference
if they get cured this year or next. Is it life threatening to
have a wart on your hand? Not in the least. A bacterial infection
is different; they can grow, doubling their numbers many times
a circadian cycle. The patient's body can't compete with the flood
of the growing bacteria, they quickly permeate and poison the
patient. Bacteria are the fastest killers among the infections
because of an overwhelming population explosion though fungal
toxins can be a thousand times more potent on a molecule to molecule
basis. The bacteria also exude a series of different toxins (that
seem to be acidic). The body's defense for both bacteria and viruses
is a fever. The bacteria have a few weak spots. All will succumb
to a rapid pH change (the stomach sanitizes food in this way),
and bacteria are extremely sensitive to the toxic fungi output,
i.e., penicillin. Clean living (detergents, bleaches), cooking
food (steamed for a given time plus pressure if possible), acid/base
swing and fungi are the quick and sure ways to defeat almost any
bacteria easily.
Let's play with the concept of the stomach eliminating the bacteria
with a pH change and see what is the predictable corollary. The
gut is under constant and unremitting assault by bacteria (as
well as by the viral and fungal microbes). A normal gut pressurizes,
in flux, by closing both valves and squeezing the contents with
enzymes and lowering the pH, i.e., making the contents very acidic.
Then it relaxes and secretes a base to neutralize the contents
or perhaps make them slightly basic, all in a short time. This
is a healthy gut system to either a chemical engineer or a doctor.
Now introduce the factor of a nervous stomach: one that constantly
dribbles acid, keeping the pH level fairly constant, day and night.
Is it the acid that eats the lining? I doubt it. Just imagine
an acid-loving bacteria finding this steady state environment.
Bacteria can even live in jet fuel, a very inhospitable environment.
This should be the first stage of an ulcer, a bacterial infestation.
Step two: fungi find the dead skin of this raw ulcer and takes
it away from the bacteria, leaving the bacteria to exist in only
the edge surrounding site as in the infection of Tinea Pedis.
Therefore to treat an ulcer of the stomach, one should use: Stage
one: a bactericide; Stage two: a bactericide with a fungicide
and get the stomach to a normal pH cycle. Stomach ulcers should
really all be treated as a Stage Two situation; that solution
handles either stage one or stage two.
Considering Schizophrenia
For the sake of this discussion, let's consider the cause of schizophrenia
to be fungal. Let's make the argument and predicate this survey
on the cycle of that mysterious illness from the viewpoint of
the illness being caused solely by fungi. We will test this hypothesis
that must cover the many idiosyncratic oddities that seem to go
with this most terrifying sickness: a mind out of the control
of its owner as if pirated by demons. What are the noted attributes
that schizophrenia is caused by fungal toxicity? What are the
indicators that this illness is even an infectious disease? All
schizophrenia is the process (chronic) schizophrenia v. reactive
(induced by external factors or learned) schizophrenia. Further,
that the cause of schizophrenia, in its earlier stages, is not
misconnected or shorted-out "wiring" (synapses firing through
faulty myelin sheaths) in the brain, but that it is of chemical
origin caused by secretions of a fungi. We will not cover the
un-learning/re-learning necessary for any recovery of any habituated
(associated reactive aspects) lifestyle caused by this illness
of process schizophrenia. Other indicators that schizophrenia
is fungal? Steroid usage is noted which is strongly pro-fungal
along with chronic nicotine, caffeine, L-dopa etc. What about
caffeine? Is caffeine possibly mildly anti-fungal? Let's look
at the molecule profile of caffeine comparing it to the Indole
(a pro-fungal molecule), and severely modified, anti-fungal, nicotine
molecules. Caffeine is a severely modified indoled molecule with
several important indole atoms exchanged for other atoms, but
retaining the indole shape. It passes the blood/brain barrier
but does not promote fungal growth: note that coffee grounds left
damp in the open sit a long time before decomposing.
A wild card thrown in at this point is an illness that could now
have a fungal implication, never heretofore considered as such:
the cardiovascular illness of myocardial infarction which is statistically
associated with schizophrenia patients. This may just be an effect
caused by the horrid medicines given, a patient's lifestyle, or
it might have something to do with a fungus. Further research
is needed. An interesting thing about medicine is the force and
volume of questions that boomerang back whenever we toss out the
smallest suggestion. What illnesses schizophrenia is associated
with provides us with a clue. LSD-25, a fungal generated toxic
indole, can give results that are indistinguishable from schizophrenia.
What other indicators do we have? Schizophrenia has a higher prevalence
in patients that have severe fungal illnesses such as Lupus, M.S.,
deficiencies in B-1, B-3 (nicotinic acid, a fungicide), prophyria
(the pyrole half of a fungi indole molecule with one type of modified-urea
attached). What is different about a fungal infection? It uses
the nitrogen offered it by fauna which it changes into a form
that the kidneys can't pass. The fungal infection is non-feverish.
Note that there is no fever ever associated with a schizophrenia
episode with one significant, unnoticed exception that of an
initial preparation of the site for a fungal infestation. If you
listen very closely, you will hear that most patients, with any
chronic illness, think it began with some other illness or mechanical
injury. This bacterial, viral or mechanical trauma initiates and
preps the site that any roving fungi (spore) infection can now
take over and dominate.
One unnoticed indicator of a fungal infestation is that it metabolizes
the urea that the human body emits to feed itself and then secretes
a modified urea that the body can't handle. Gouty arthritis is
a good example. Note that a fungus on a topical surface such as
Irritable Bowel Disease, psoriasis, sinusitis, etc., can drain
into a void and not cause toxicity to the body due to its inability
to be processed and pass on to the kidneys. So if there is a fungus
causing schizophrenia where is the modified-urea that the body
can't handle? To answer the modified-urea question we must know
where the fungal infection is sited. Since the fungi are so potent,
only a very few are needed. In fact such a small number of fungi
are needed that they and the modified-urea could escape any detection
other than an implied existence. Since the hypothetical toxic
fungus indole secretion can pass the blood/brain barrier, the
fungal infestation could even be under that patient's toenails
and the fungi's extreme cell-wall penetrating toxins could find
their way to the most sensitive of all human organs, the brain.
Not knowing where the fungi is sited or the size and number involved,
hence the place and volume of the fungi-secreted modified-urea,
makes looking for the fungal site impossible. In gouty arthritis
the fungal site is obvious. There is a hint in the voluminous
medical literature to show where we should look if we prescribe
a small dose of logic for ourselves. The blood/brain barrier should
stop most fungi. The closer the fungal site is to the brain, the
more effective an unnoticeably small number of fungi would be.
Don't forget, fungi-like steroids and cortisone was first culled
from the outside surface of the brain. If the blood/brain barrier
wasn't there, neither would be the brain, it would be covered
with fungi and putrefy and mortify like a rotting log. This concept
would put the fungal site at or near the stalk of the brain just
outside the blood/brain barrier. The exceedingly small infestation
would now produce such a small amount of modified-urea as to be
unnoticed. The thermographs of schizophrenia patients show lesser
activity at most sites of the brain, but at the blood/brain barrier,
more activity or heat is indicated. That wasn't so hard was it?
It is a point to first look for a fungus. Note that one of the
characteristics of some severe fungal infections is that the patient
never gets a cold. We know fungi provide protection from bacterial
infections but there is also some protection from viruses too.
Why, I don't have a clue. What provokes a fungal growth, hence
schizophrenic episode? Lactic acid seems to be a provocateur par
excellence. When I had my irritable bowel disease, extreme muscle
work caused the illness to get very severe. Schizophrenia patients
given a lactic acid injection get schizophrenic episodes: normal
people do not. This is a definitive chemical test for a very hard-to-define
illness. When schizophrenic patients are on the standard schizophrenia
medicine, they do not have these attacks with lactic acid injections.
Lactose sugar is another fungal activator or provocateur (pill
binder is made out of milk and not to be confused with lactic
acid). Have you ever heard of a woman getting yeast infection
after being treated with a strong dose of one of the penicillins?
This is very common. What causes a steroid imbalance in the human
body? Stress, puberty, prÑ&Mac173;@
Another anti-fungal is iodine (as noted, it seems to be anti-viral
also) but much weaker and milder than chloride as an anti-fungal.
Iodine is a powerful anti-fungal (and in what seems to be higher
doses, also anti-bacterial). I consider its reduction below the
RDAs as a proposed cause of a higher rate of fungal infections
like schizophrenia, asthma, IBD, arthritis, lupus, etc. Modern
day dietary reduction of NaCl with iodine is noted also. What
other vitamin chemistry promotes fungal infections? Vitamin A
is a steroid imitator. Bacterial growth and subsequent demise
prepares the sites of the body for further fungal infection. Where
a bacteria can co-exist with fungi in a patient, these bacteria
can cause great and rapid damage to the patient as in Mega-colon
in the ulcerative colitis patient. How about alcohol? What should
its theoretical effect be to a schizoid patient? As always, let
us go to the source. The common source of alcohol (methyl carbinol)
is yeast, a major subdivision of fungi. Alcohol is anti-bacterial
and because it comes from a fungus it is therefore pro-fungal.
Alcohol blood dilution (drunkenness) provides a temporary calming
effect on the schizoid patient. Because any bacteria in contact
with alcohol is suppressed, the rebound growth is increased for
the fungi which intensifies the future episode of schizophrenic
illnesses. The greater the fungal infestation, the more devastating
the schizophrenic episode: alcoholism is associated with schizophrenia.
The anti-fungal chemicals used to fight schizophrenia are indicators
as to the cause of this terrifying disease. One of a galaxy of
the tri-cyclines is #2186, chlorpromazine. Miscellaneous other
near tri-cyclines only slightly less related to the molecule #7220
is carbamazepine (#1783), a tri-cycline with an extra atom in
the center ring. We must look at the root of any chemical to get
a handle on its function and use. Also look at mercurochrome (Merbromine
#5757) which is interesting because it at least provides a little
variety in the repetitive tri-cyline theme by using the old standby
mercury (Hg). In a micro/micro dose, Hg can be very effective
but long term use can be devastatingly toxic. To be effective,
it has to kill something, so Hg is called dangerous. Misused it
surely is, but is it safe to stay ill? So Hg based medicines are
called a health hazard (patient rights have expired) and lets
a drug company make money with the new chemicals that don't work
near as well. How else can the new chemicals make more profit?
The price is higher just because only the patent holder can issue
a permit to make them. Also, it takes more than one drug to do
the job and many more treatments, and the new drugs which have
horrid side effects, require a doctor's prescription and now the
still ill patient has to spend more time in the hospital. Medication,
hence cure, that should cost a dime now costs tens of thousands
of dollars and even the life of the patient. Note that young children
who get IBD's have a 30% to 40% mortality rate by the time they
reach the age of majority. This is when they receive the "Best
of the FDA/AMA's Medical Care." This just shows the seriousness
of the illness. It takes only one dollar of chemicals to cure
each child, which cannot be administered within the FDA/AMA guidelines.
The profit motive cannot help but make the most sincere doctors
prescribe expensive drugs and further tests (just to be sure the
patient is safe). If a doctor fails to play this game, the general
microbe being inferred indirectly, then that doctor is liable
to feed the vultures who really regulate medicine and the FDA/AMA.
Because the precisely correct and approved chemical trade name
(active patent) will be spelled very differently, though the chemicals
will show 90% commonality and really be interchangeable, a doctor
will be Keel-hauled by our judicial system to teach him a lesson
in the importance of greed to our GNP. As the Japanese say, "The
high nails will be driven down." This is done at the cost of multiplying
the danger to the patient and the cost to the general public.
The molecule derivatives of the Tri-C's are damaging to the liver
whereas the nicotine is not. The advantage of the tri C's is due
to the strong numbing or blocking effect on the brain cells which
seems to be needed in many schizophrenic episodes. This gain is
often at the expense of severe and even fatal damage to the liver
in long term tri-C usage. The knowledge of what is really going
on can help the doctor/patient minimize the use of the dangerous
but necessary tri-C's.
On examination of schizophrenic episodes we find the following
occurrence repeatedly: severe stress, a 14-day wait for the episode
to occur (10 to 12 days for the aura?). How does our model of
a fungal infection fit into this? We need to find out how long
it takes a spore to mature to an active fungus in the human body
to answer that question. Steroids strongly promote fungal growth.
Stress releases a massive amount of steroids. If these steroids
are not metabolized or used in the body, the fungi use them and
grow, then when their growth is not sustained, die and release
their toxins. The toxins of fungi seem to be based on variations
of the molecule #4869, the indole molecule. The most notorious
example of this indole base is the molecule #5507 (LSD-25) which
is made by a fungus originally. It so happens that a person on
LSD-25 can be indistinguishable from a schizophrenia patient suffering
a schizophrenic episode.
Nicotine has the virtue of metabolizing into vitamin B-3, which
is noted as a chemical to treat schizophrenia. Note that many
schizophrenic patients show a calming of episodic events while
smoking. This use of nicotine allows a schizophrenia patient to
focus. Isn't this strange, a stimulant calming a person? And that
stimulant just happens to be anti-fungal? And the other chemicals
used to treat schizophrenia just happen to be anti-fungal also?
There are some other odd illnesses that seem to be prevented by
or respond to anti-fungal agents such as nicotine and the tri-cyclines.
Nicotine users have been noted to have less Alzheimer's, Parkinson's,
Sleep apnea, IBD's, Asthma's, etc. by Dr. Jarvik. A strong argument
can be made for each of the foregoing illnesses to be caused by
a low or non-feverish fungal infection.
In "Menke's Kinky Hair" syndrome the body cannot absorb copper.
A trace of copper, a noted fungicide, cures the problem. That
is it stops the progress of the illness, which is in the brain,
but doesn't repair any brain damage. Doesn't Cu uptake get reduced
by alcohol? Don't people with arthritis (a fungal illness) often
swear by copper bracelets? Certain things that have been noted
about viral infections are that a "one pill cure" or "one serum
injection" is possible. This has led many to think that all illnesses,
even if they are not a virus can be cured with one pill. The treatment
of a bacterial infection in just a few days (commonly ten days)
has bolstered this "pipe dream" that a single, correct pill will
cure "it," whatever "it" is, even though constant cleanliness
and preventive measures are really needed, i.e., a "defensive
lifestyle." Hence limiting bacterial infections is really a constant
lifestyle of refrigeration, washing, eating right, chlorinated
water, and good habits in general.
Fighting a fungus is an ongoing battle or a "defensive lifestyle"
too, in the pursuit of good health. Gouty arthritis is a splendid
example of long-term management that is effective only with clear
insight to fungal treatment and schizophrenia is an obvious and
excellent implied infection of fungi whose treatment is using
anti-fungal measures with no pro-fungi slip-ups which cannot be
done without a broad concept of the cause. The past treatment
of this serious illness is strictly trial and error without any
overall concept of what is going on as though there was no broad
concept available or even hinted at by the chemistry involved.
Those that propose a yeast cause are on track though and it may
well be a yeast that is the causative microbe, though the other
fungi should still be open for consideration. Treatment without
logic schema (compass), is treatment founded on a data base which
is now quickly lost in such a fog as to be virtually useless (medicine
then becomes a lifeboat without a rudder for which the hull is
missing.) This style of treatment totally avoids any unifying
theme hence any real chance of a cure due to the lack of reasonable
and/or proper diagnoses. A patient or doctor cannot go in more
than one direction at once and succeed. Even if one proposes the
wrong cause, genetic, chemical, external, microbial, etc., at
least the trial and error method will either prove or disprove
the advanced theory permitting its refinement or erasure. Most
of today's treatments are more political and seem to lack any
solid scientific basis. A scientific basis is where each concept
is firmly attached and proved by known scientific cause and effect
to a previous established point, making a chain connecting the
start to the finish; the cause to the cure. The point of the beginning
is always the simple sciences: first year chemistry, first year
biology, first year zoology, etc. In the illness of chronic or
process schizophrenia, we suggest the cure to the physical cause
to be anti-fungal regimens which can use all the existing anti-schizophrenia
chemicals to treat fungi. These medicines prescribed should all
be anti-fungal, ie., nor-nicotine and nicotine (very limited usage),
Vitamin B-1 through B-6, potassium and lithium, iodine and other
halogens, sulfates and sulfur, iron supplements, etc. (copper
ions should be included too but I don't know which ones are safe
and in what doses.) The chemicals that must be avoided or severely
limited are the pro-fungal vitamin A, B-12, D's and K's. Also
avoid the strongly pro fungal pill binder, lactose and any milk
products (excepting hard cheeses), and in particular the chlorophylls
must be avoided. Heavy or even modest physical workouts must be
avoided because they generate lactic acids at a rate that the
body cannot handle. This regimen, which uses most of the existing
schizophrenic treatments with only slight modification, should
be easy, safe and provide results quickly to prove or disprove
this contention of a fungi being the cause of schizophrenia.
Correspondence:
David Barnes
215 Christine Dr.
San Pablo, California 94806 USA
510-223-1592
Fax 510-222-1650
Email: DaveyHugh@aol.com
Letters
Infantile Autism, Diet & Schizophrenia
Editor:
In November of 1996, my son David, a three year-old child, was
diagnosed with moderate to severe early infantile autism. We immediately
realized that milk irritated our child's autistic condition. Within
three weeks of removing milk from our son's diet, we noticed an
improvement in his eye contact and socialization skills, as well
as a decrease in self stimulating behaviors (i.e. spinning and
hand flapping) and self abusive behaviors such as head banging.
We also removed wheat from our son's diet. However, David's progress
was staggered, and we would eliminate one troubling behavior from
his repertoire via behavioral therapy, only to find another replacing
it. Holding a BA in behavioral psychology, I felt that something
was missing, a key that I hadn't determined. Relentless poring
over every resource in this century regarding autism, I became
interested in the connection between autism and systemic yeast
infection. I was prepared to start my son on Nystatin treatment
when I encountered Mr. David Barnes on line. Mr. Barnes recommended
an unusual eating plan designed to remove the yeast from my autistic
David's system. Yes, I was skeptical, but I also knew that Nystatin
treatment in itself could have adverse side effects for my son.
David Barnes recommended removing milk, dairy products, wheat,
yeasts and raw vegetables from my son's diet. He also advised
that we remove the fungi/yeast from his diet that his pediatrician
had suggested for nutrition. Mr. Barnes also advised to boil all
vegetables in salt (NaCl) water before cooking. This interested
me, because my son has an intense craving for table salt. An innate
attempt of the body to self medicate, perhaps? Mr. Barnes also
advised the addition of dietary copper and a dietary iodine, but
in minimal dosages, having noticed that most auto-immune illnesses
have an identified enzyme that depletes body copper.
I followed his advice, with no other changes in my child's routine.
Within three days, my son slept through the night for the first
time in his life. Within three weeks, my child was spontaneously
requesting and giving affection...even an occasional kiss. He
started to play with his toys in a more appropriate manner, stacking
blocks and putting small objects inside of larger ones. Most interesting,
my previously diagnosed three year-old child began crawling, a
critical developmental stage he had skipped. Six weeks into David
Barnes' recommended new eating program, our autistic child began
exploring my face, eyes, ears, nose and mouth. He now follows
simple commands, and occasionally uses a word in context. My son
is a happy child, hardly recognizable from the wandering ghost
he was only eight weeks ago.
My son will begin tomorrow, a program for language-delayed toddlers
that he did not qualify for six months ago. His condition was
considered too severe. Will my son recover from autism? I don't
know. But I do know that David Barnes' advice, applied with the
conservatism of a sheltering mother, changed my little boy's life.
And mine as well. I have used David Barnes' anti-fungal diet also,
and the arthritis in my feet seems to be in total remission.
Traci L. Yates-Poff
Grateful Mother
StarMuser@aol.com
Addendum from David Barnes
The child then grew rapidly and a year later is now 4 and ahead
of his age group in many areas, though somewhat slow in speech.
He is using full sentences in context and with clear meaning and
composing sentences and plays with other children and likes to
play games. He has a pet dog that he cares for.
At the age of three (3) he had never spoken and often sat there
and banged his head against the wall in destructive behavior.
Now he goes to school and is helpful to his mother and a joy to
all. Most of his behavior changed in only 10 days or so. Progress
was sure and leveled off after that but was still ahead of rate
of improvement for his peers. His weight was that of his younger
sister when this began, a year later he weighs close to his older
sister.
Editor's Comment:
The theory and discussion regarding an anti-fungal treatment for
autism referred to an article on "Schizophrenia: A Proposed Cause
and Cure" by David Barnes, published in the June, 1994 issue of
the TLfDP (#131).